Augustyniak A, Czy?ewska-Dors E, ?agowski D, Domań. Clinical Course, Laboratory Findings, and Tissue Distribution of Highly Pathogenic Avian Influenza A(H5N1) Virus in a Naturally Infected Domestic Cat. Viruses. 2026; 18(9):1031. Abstract submitted by kickingbird at 1 minute(s) ago from Viruses. 2026; 18(9):1031 In June 2023, Poland experienced an unprecedented outbreak of highly pathogenic avian influenza (HPAI) H5N1 virus infections in cats, raising concerns about the role of cats in HPAI H5N1 virus ecology ... Gabiden A, Mutaliyeva A, Usserbayev A, Yermekbayev. Antigenic and Molecular Genetic Evolution of Influenza Viruses in Kazakhstan from 2017 to 2025. Biology. 2026; 15(18):1640. Abstract submitted by kickingbird at 1 minute(s) ago from Biology. 2026; 15(18):1640 Influenza viruses continuously evolve, which can change their circulation patterns and reduce recognition by existing immunity and vaccine-induced antibodies. Continuous surveillance is therefore important ... Antoni Wrobel, etc.,al. [preprint]H18 haemagglutinin binds MHC class II at a site distant from the canonical sialic-acid binding site. https://doi.org/10.21203/rs.3.rs-10549591/v1. Abstract submitted by kickingbird at 6 minute(s) ago from https://doi.org/10.21203/rs.3.rs-10549591/v1 Influenza virus haemagglutinin (HA) binds sialic-acid receptors to mediate cell entry of canonical influenza A viruses (IAVs) and is a key determinant of viral tropism and host range. However, the bat-derived ... Alanis Silva Melgarejo, etc.,al. [preprint]Highly Pathogenic Avian Influenza Clade 2.3.4.4b in Antarctic Pinnipeds: first evidences of virus circulation in Weddell seals. https://doi.org/10.21203/rs.3.rs-10922875/v1. Abstract submitted by kickingbird at 7 minute(s) ago from https://doi.org/10.21203/rs.3.rs-10922875/v1 The ongoing expansion of highly pathogenic avian influenza virus (HPAIV) H5N1 clade 2.3.4.4b has resulted in unprecedented outbreaks among wild birds and marine mammals, raising concerns about viral dissemination ... Juliane Lang, etc.,al. [preprint]Compensatory Mutations in Avian Influenza H7N1 Hemagglutinin Drive Avian Host Adaptation and Antigenic Drift without Enhancing Replication in Human Cells. https://doi.org/10.21203/rs.3.rs-10351951/v1. Abstract submitted by kickingbird at 9 minute(s) ago from https://doi.org/10.21203/rs.3.rs-10351951/v1 Avian influenza A viruses (AIVs) of the H7 subtype have caused outbreaks in poultry and human ranging from mild to fatal disease. In Europe, H7 viruses have demonstrated an unusual capacity for prolonged ... |